What Other Diseases "Masquerade" as Rheumatoid Arthritis? Part 1 - The Non-Infectious Group

Rheumatoid arthritis (RA) is the most common"seronegative" types of arthritis. The term
form of inflammatory arthritis and affects more'seronegative' means that testing for rheumatoid
than 2 million Americans. The diagnosis is not easyfactor is negative. Symptoms of adult SAs
to make in many instances. There are more thaninclude:o Back and/or joint pain;o Morning
100 different kinds of arthritis. Most of themstiffness;o Tenderness near bones;o Sores on the
involve inflammation. When a patient goes to askin;o Inflammation of the joints on both sides of
rheumatologist to get a diagnosis, there is athe body;o Skin or mouth ulcers;o Rash on the
process of elimination in order to arrive at thebottom of the feet; ando Eye inflammation.
proper diagnosis. This process of elimination isOccasionally, arthritis similar to that seen in RA
called "differential diagnosis."can be present. Careful history and physical
Differential diagnosis can be a difficult undertakingexamination can often distinguish between these
because so many forms of arthritis, particularlyconditions, especially if an obvious disease that is
inflammatory forms of arthritis look alike.promoting inflammation is present (psoriasis,
Generally it is helpful to divide the differentialinflammatory bowel disease, etc.). In addition, RA
diagnosis of rheumatoid arthritis into two groups.rarely affects the DIP joints- the last row of
The first group are the non-infectious diseases tofinger joints. If these joints are involved with
consider and the second group are theinflammatory arthritis, the diagnosis of an SA is
infection-related conditions.possible. (Note of caution: a condition known as
Since the discussion is rather long I have choseninflammatory erosive nodal osteoarthritis can also
to divide the article into two parts.affect the DIP joints). RF and anti-CCP antibodies
The following is a partial list of forms ofare negative in SAs, although, rarely, in cases of
inflammatory arthritis that can be seen and mustpsoriatic arthritis there may be elevations of RF
be considered when evaluating a patient withand anti-CCP antibodies.
inflammatory symptoms of arthritis and are notGout is caused by deposits of monosodium urate
infection related.(uric acid) crystals into a joint. Gouty arthritis is
RA is an autoimmune chronic inflammatoryacute in onset, very painful, with signs of
disease, primarily involving the peripheral jointssignificant inflammation on exam (red, warm,
(hands, wrists, elbows, shoulders, hips, knees,swollen joints). Gout can affect almost any joint in
ankles, and feet). It can also affect non jointthe body, but typically affects cooler areas
structures such as the lung, eye, skin, andincluding the toes, feet, ankles, knees, and hands.
cardiovascular system.Diagnosis is made by drawing fluid from an
RA may start slowly with nonspecific symptoms,inflamed joint and analyzing the fluid.
including fatigue, malaise (feeling "blah"), appetiteDemonstrating monosodium urate crystals in the
loss, low-grade fever, weight loss, and vague jointjoint fluid is diagnostic, although finding elevated
pains, or it may have an explosive onset withserum levels of uric acid can also be helpful.
inflammation involving multiple joints. The jointIn most cases, gout is an acute single joint
symptoms usually occur bilaterally- both sides ofdisease that is easy to distinguish from RA.
the body equally involved- and symmetric.However, in some cases, chronic erosive joint
Erosions- damage to the joint- can be seen withinflammation where multiple joints are involved
x-ray. In about 80% of cases, elevated levels ofcan develop. And, in cases where tophi (deposits
rheumatoid factor (RF) or anti-cyclic citrullinatedof uric acid) are present, it can be difficult to
antibodies (anti-CCP) are present in the blood.distinguish from erosive RA. However, crystal
There appears to be a correlation between theanalysis of joints or tophi and blood tests should
presence of anti-CCP antibodies and erosions.be helpful in distinguishing gout from RA.
Juvenile rheumatoid arthritis (JRA) occurs inCalcium pyrophosphate deposition disease (CPPD),
children under the age of 16. Three forms of JRAalso known as pseudogout, is a disease is caused
exist, including oligoarticular (1-4 joints),by deposits of calcium pyrophosphate dihydrate
polyarticular (more than 4 joints), andcrystals in a joint. The presence of these crystals
systemic-onset or Still's disease. The latterin the joints leads to significant inflammation.
condition is associated with systemic symptoms --Establishing the diagnosis includes using:o Detailed
including fever and rash in addition to joint disease.medical history;o Withdrawing fluid from a joint to
Polyarticular JRA has similar characteristics to adultcheck for crystals;o Joint x-rays to show crystals
RA. It causes about 30% of cases of JRA. Mostdeposition in the cartilage (chondrocalcinosis); ando
children with polyarticular JRA are negative for RFBlood tests to rule out other diseases (e.g., RA or
and their prognosis is usually good.osteoarthritis).
Approximately 20% of polyarticular JRA patientsIn most cases, CPPD arthritis presents with single
have elevated RF, and these patients are at riskjoint inflammation. In some cases, CPPD disease
for chronic, progressive joint damage.can present with chronic symmetric multiple joint
Eye involvement in the form of inflammation-erosive arthritis similar to RA. RA and CPPD
called uveitis- is a common finding in oligoarticulardisease can usually be told apart by joint
JRA, especially in patients who are positive foraspiration demonstrating calcium pyrophosphate
anti-nuclear antibody (ANA), a blood test that iscrystals, and by blood tests, including RF and
often used to screen for autoimmune disease.anti-CCP antibodies, which are usually negative in
Uveitis may not cause symptoms so carefulCCPD arthritis. A complicating feature is that RA
screening should be performed in these patients.and CPPD can coexist!
SLE is an inflammatory, chronic, autoimmuneSarcoidosis is an inflammatory joint disorder. The
disorder that can involve the skin, joints, kidneys,majority of patients with this disease have lung
central nervous system, and blood vessel walls.disease, with eye and skin disease being the next
Patients may present with 1 or more of themost frequent signs of disease. Although the
following: butterfly-shaped rash on the face,diagnosis of sarcoidosis can be made on clinical
affecting the cheeks; rash on other parts of theand x-ray presentation alone, sometimes the use
body; sensitivity to sunlight; mouth sores; jointof tissue biopsy with the demonstration of
inflammation; fluid around the lungs, heart, or"noncaseating granulomas" is necessary for
other organs; kidney abnormalities; low whitediagnosis.
blood cell count, low red blood cell count, or lowArthritis is present in 15% of patients with
platelet count; nerve or brain inflammation;sarcoidosis, and in rare cases can be the only sign
positive results of a blood test for ANA; positiveof disease. In acute sarcoid arthritis, joint disease
results of a blood test for antibodies tois usually of rapid onset. It is symmetric involving
double-stranded DNA or other antibodies.the ankles, although knees, wrists, and hands can
Patients with lupus can have significantbe involved. In most cases of acute disease, lung
inflammatory arthritis. As a result, lupus can beand skin disease are also present. Chronic sarcoid
difficult to distinguish from RA, especially if otherarthritis can be difficult to distinguish from RA.
features of lupus are not present. Clues thatAlthough RA-specific blood tests, such as RF and
favor a diagnosis of RA over lupus in a patientanti-CCP antibodies, can be helpful in distinguishing
presenting with arthritis affecting multiple jointsRA from sarcoidosis, in some cases a biopsy of
include lack of lupus features, erosions (jointjoint tissue may be required for diagnosis.
damage) seen on x-rays, and elevations of RFPolymyalgia Rheumatica (PMR) is a disease that
and anti-CCP antibodies.leads to inflammation of tendons, muscles,
Polymyositis (PM) and dermatomyositis (DM) areligaments, and tissues around the joints. It
types of inflammatory muscle disease. Thesepresents with large muscle pain, aching, morning
conditions typically present with bilateral (bothstiffness, fatigue, and in some cases, fever. It can
sides involved) large muscle weakness. In thebe associated with temporal arteritis (TA), also
case of DM, rash is present. Diagnosis consists ofknown as giant-cell arteritis, which is a related but
finding the following: elevation of muscle enzymemore serious condition in which inflammation of
levels in the blood [the two enzymes that arelarge blood vessels can lead to blindness and
measured are creatine kinase (CPK) and aldolase],aneurysms. Also, a peculiar syndrome where use
signs and symptoms, electromyograph (EMG)- anof the arms and legs leads to cramping because
electrical test- alteration, and a positive muscleof insufficient blood flow (limb claudication) can
biopsy.occur. PMR is diagnosed when the clinical picture is
In addition, in many cases abnormal antibodiespresent along with elevated markers of
specific for inflammatory muscle disease can beinflammation (ESR and/or CRP). If temporal
elevated.arteritis is suspected (headache, vision changes,
In both PM and DM, inflammatory arthritis can belimb claudication), biopsy of a temporal artery
present and can look like RA. Both inflammatorymay be necessary to demonstrate inflammation
muscle disease and RA can affect the lungs. Inof blood vessels.
RA, muscle function will usually be normal. Also, inPMR and TA can present with symmetric
PM and DM, erosive joint disease is unlikely. RFinflammatory arthritis similar to RA. These
and anti-CCP antibodies are typically elevated indiseases can usually be distinguished by blood
RA but not PM or DM.testing. In addition, headaches, vision changes, and
SAs -- psoriatic arthritis, reactive arthritis,large muscle pain are uncommon in RA, and if
ankylosing spondylitis, and enteropathic arthritis --these are present, PMR and/or TA should be
are a category of diseases that cause systemicconsidered.
inflammation, and preferentially attack parts ofIn part 2 of this article, I will discuss infectious
the spine and other joints where tendons attachdiseases that need to be considered in the
to bones. They also can cause pain and stiffnessdifferential diagnosis of rheumatoid arthritis. When
in the neck, upper and lower back, tendonitis,RA is suspected, it is critical to consult with an
bursitis, heel pain, and fatigue. They are termedexpert rheumatologist.